The theory that China obtained the coronavirus via a Canadian research program, and started molding it into a bioweapon at the Institute of Virology in Wuhan before it somehow escaped could be an attempt by the establishment (the Davosocracy,) to spread fear and panic as they see resurgent nationalism across the developed world and growing scepticism about the Greta Thunberg fronted campaign to reignite fear and panic about runaway climate change causing millions of deaths and widespread destruction. Establishment controlled news media such as The New York Times, The Guardian, CNN and the BBC have dismissed the claim as ‘fake news’, though the story was widely shared across independent-leaning media.
According to the weaponised virus theory the virus, which was developed by infectious disease experts may have originated in the Wuhan-based lab of Dr. Peng Zhou, China’s leading researcher on bat immune systems (whoda though there were actually people out there making a living from researching the immunse systems of bats?) who has been studying in what ways the flying rodents’ immune systems respond to the presence of destructive viruses. The theory suggests that eith by accident or design the virus was carried out of the lab, and somehow ended up in animals and fish on stalls in the Wuhan wet market. Claims that the market is where the virus supposedly originated by jumping species from bats being sold for food to other species have been dismissed as a conspiracy theory.
Now, a respected researcher, Dr Prashant Pradhan, is leading a team looking into how epidemics spread, and who recently came in for a lot of criticism by tweeting that the virus appeared to be much more contagious than was originally thought, is pointing out that anomalies in SARS – CoV2 virus’s genome suggest it may be a genetically engineered organism for use as a biological weapon.
Specific inserts in the 2019-nCoV spike protein to HIV-1 gp120 have rendered Indian researchers baffled by parts of the virus’s RNA that bear no relation to other coronaviruses strains like SARS, and instead appear to be closer to HIV. The virus has even been shown to be susceptible to HIV medications.
A link to the full paper (.pdf) is provided below, like most scientific papers it is heavy going for the lay person, but those with the patience to read it will find the first part focuses on the unique nature of 2019-nCoV, and observes that four amino acid sequences in the Wuhan Coronavirus are homologous to amino acid sequences in HIV1.
Why do the authors think the virus may be man-made? Because, they say, when looking at the above insertions which are not present in any of the closest coronavirus families, “it is quite unlikely for a virus to have acquired such unique insertions naturally in a short duration of time.” Instead, they can be found in cell identification and membrane binding proteins located in the HIV genome.
READ THE FULL REPORT in .pdf format
For other readers, here are a few key points from the:
… To further investigate if these inserts are present in any other corona virus, we performed a multiple sequence alignment of the spike glycoprotein amino acid sequences of all available coronaviruses (n=55) [refer Table S.File1] in NCBI refseq (ncbi.nlm.nih.gov) this includes one sequence of 2019-nCoV[Fig.S1]. We found that these 4 insertions [inserts 1, 2, 3 and 4] are unique to 2019-nCoV and are not present in other coronaviruses analyzed. Another group from China had documented three insertions comparing fewer spike glycoprotein sequences of coronaviruses .
We then translated the aligned genome and found that these inserts are present in all Wuhan 2019-nCoV viruses except the 2019-nCoV virus of Bat as a host [Fig.S4]. Intrigued by the 4 highly conserved inserts unique to 2019-nCoV we wanted to understand their origin. For this purpose, we used the 2019-nCoV local alignment with each insert as query against all virus genomes and considered hits with 100% sequence coverage.
Surprisingly, each of the four inserts aligned with short segments of the Human immunodeficiency Virus-1 (HIV-1) proteins. The amino acid positions of the inserts in 2019-nCoV and the corresponding residues in HIV-1 gp120 and HIV-1 Gag are shown in Table 1.
When looking at the above insertions which are not present in any of the closest coronavirus families, “it is quite unlikely for a virus to have acquired such unique insertions naturally in a short duration of time.” Instead, they can be found in cell identification and membrane binding proteins located in the HIV genome.
Since the S protein of 2019-nCoV shares closest ancestry with SARS GZ02, the sequence coding for spike proteins of these two viruses were compared using MultiAlin software. We found four new insertions in the protein of 2019-nCoV- “GTNGTKR” (IS1), “HKNNKS” (IS2), “GDSSSG” (IS3) and “QTNSPRRA” (IS4) (Figure 2). To our surprise, these sequence insertions were not only absent in S protein of SARS but were also not observed in any other member of the Coronaviridae family (Supplementary figure). This is startling as it is quite unlikely for a virus to have acquired such unique insertions naturally in a short duration of time.The insertions were observed to be present in all the genomic sequences of 2019-nCoV virus available from the recent clinical isolates. To know the source of these insertions in 2019-nCoV a local alignment was done with BLASTp using these insertions as query with all virus genome. Unexpectedly, all the insertions got aligned with Human immunodeficiency Virus-1 (HIV-1). Further analysis revealed that aligned sequences of HIV-1 with 2019-nCoV were derived from surface glycoprotein gp120 (amino acid sequence positions: 404-409, 462-467, 136-150) and from Gag protein (366-384 amino acid) (Table 1). Gag protein of HIV is involved in host membrane binding, packaging of the virus and for the formation of virus-like particles. Gp120 plays crucial role in recognizing the host cell by binding to the primary receptor CD4.This binding induces structural rearrangements in GP120, creating a high affinity binding site for a chemokine co-receptor like CXCR4 and/or CCR5.
We are not qualified to comment on the quality of this work beyond saying that evidence is stacking up that points to there being something extremely dodgy about the origins of the coronavirus currently running wild in China. Because us old farts have surprisingly good memories we are reminded of the Ebola outbreak in west Africa a few years ago, which again was reported to be a previously unknown strain of the virus, much more infectious than those identified in previous outbreaks.
More Evidence Deadly Ebola Strain Was Created By US Biological Weapons Research Scientists
from Russ Winter‘I presented the case for the Kemena bio-lab as the source of spreading the Ebola outbreak in Saturday’s post, and I had to circle back and debunk the debunkers. It states on the hospital consortium’s own website that it is involved in research on lethal diseases at Kenema.
Indeed, starting in January, the consortium running the Kenema lab inked a $140 million deal with Department of Defense and a pharma company called Tekmira to conduct Phase I Ebola vaccine trails at Kenema on humans [see “Tekmira Doses First Subject in Human Clinical Trial of TKM-Ebola” press release].
The information provided begs the question: Where did they get the human subjects? There was no outbreak as of the end of March in Sierra Leone. Seems the DoD/pharma goon squads were recruiting Ebola trial participants from nearby Guinea, where there was a small outbreak underway.’
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